A nurse is assessing a patient with type 1 diabetes mellitus… | 마이메르시 MyMerci
Adult Health
문제
A nurse is assessing a patient with type 1 diabetes mellitus who was brought to the emergency department. Which assessment finding would be the priority concern?
A 28-year-old patient with type 1 diabetes mellitus is brought to the emergency department by family members who report the patient has been vomiting for 2 days and appears confused.
1Blood glucose level of 180 mg/dL
2Mild dehydration with decreased skin turgor
3Fruity odor on breath with deep, rapid respirations✓ 정답
4Complaint of abdominal pain and nausea
해설
The fruity odor on breath with deep, rapid respirations (Kussmaul respirations) is the most critical assessment finding that indicates diabetic ketoacidosis (DKA). This represents the body's compensatory mechanism to eliminate excess acids through respiratory alkalosis.
심화 해설
Core Nursing ExplanationKey Concept Analysis: This question tests the nurse's ability to recognize a life-threatening complication of Type 1 Diabetes Mellitus (T1DM) and prioritize assessment findings. The core theme is identifying Diabetic Ketoacidosis (DKA). DKA is a metabolic crisis caused by a severe insulin deficiency, leading to hyperglycemia, ketone production, and metabolic acidosis. The body attempts to compensate for the acidosis by exhaling more carbon dioxide, resulting in a distinctive respiratory pattern.
Answer Rationale: Key Point! Option ③, "Fruity odor on breath with deep, rapid respirations," is the priority finding because it is a classic, direct sign of Kussmaul respirations and ketosis. The fruity odor is from acetone (a ketone body) expelled via the lungs, and the deep, rapid breathing (Kussmaul respirations) is the body's attempt to blow off excess acid (CO2) to correct the metabolic acidosis. This finding indicates a severe, decompensated state requiring immediate intervention to prevent shock, cerebral edema, or death.
Distractor Analysis:
Watch out for confusion! Option ①: A blood glucose of 180 mg/dL is elevated, but it is not critically high for a patient with diabetes and is not diagnostic of DKA on its own. In DKA, glucose is often much higher (e.g., > 250 mg/dL). More importantly, the glucose level alone does not indicate the presence of the life-threatening acidosis.
Option ②: Mild dehydration is a concern and a common finding in DKA due to osmotic diuresis from hyperglycemia. However, it is a consequence of the underlying problem. The respiratory signs point directly to the cause (acidosis), which is the more immediate threat to physiological stability.
Option ④: Abdominal pain and nausea are common symptoms of DKA but are non-specific. They could be caused by many other conditions (e.g., gastroenteritis). They do not, by themselves, signal the severity of the metabolic derangement like Kussmaul respirations do.
Related Concepts: The nursing priority follows the ABCs (Airway, Breathing, Circulation) framework. While dehydration affects circulation, the abnormal breathing pattern is a direct threat to the body's acid-base balance and oxygenation, making it the top priority. Always assess for Kussmaul respirations and ketone odor in any diabetic patient with illness or altered mental status.
Concept Summary
Concept
Key Points
Diabetic Ketoacidosis (DKA)
Life-threatening complication of insulin deficiency. Hallmarks: Hyperglycemia, Ketosis, Metabolic Acidosis.
Kussmaul Respirations
Deep, rapid, sighing breaths. The body's compensatory mechanism (respiratory alkalosis) for metabolic acidosis.
Priority Assessment in DKA
Airway/Breathing (Kussmaul), then Circulation (dehydration, hypotension), then neurological status (confusion).
Severe insulin resistance, Extreme dehydration, No significant acidosis
Excess insulin or oral hypoglycemics, Low blood glucose
Blood Glucose
High (>250 mg/dL)
Very High (>600 mg/dL)
Low (
임상 시나리오
Nursing Clinical Practice GuideClinical Scenario: You are the triage nurse in the ED. A 28-year-old with known T1DM is brought in by family. They report 2 days of vomiting, polyuria, and now confusion. The patient is lethargic, skin is warm and dry, and you detect a distinct fruity smell as you lean in to assess their airway.
Nursing Intervention Strategy:
Immediate Assessment (ABCs): Ensure patent airway. Assess breathing rate, depth, and pattern (Kussmaul). Attach pulse oximeter and cardiac monitor. Obtain vital signs, noting hypotension and tachycardia from dehydration.
Critical Actions: Administer oxygen via nasal cannula if indicated. Establish two large-bore IV lines. Draw stat labs: Blood glucose, Basic Metabolic Panel (BMP), Venous Blood Gas (VBG) or Arterial Blood Gas (ABG), serum ketones, and urinalysis for ketones and glucose.
Collaborative Treatment: Anticipate and initiate orders per DKA protocol: Aggressive IV 0.9% NaCl infusion, IV insulin drip (starting at 0.1 unit/kg/hr), and potassium replacement once levels are known and urine output is confirmed.
Ongoing Monitoring: Monitor blood glucose hourly. Monitor vital signs, neurological status, and urine output closely. Watch for signs of cerebral edema (sudden headache, change in neuro status) – a rare but deadly complication, especially during rapid correction in younger patients.
Patient Safety and Precautions:
IV Insulin Drip: Never administer IV insulin as a bolus for DKA. It must be given as a continuous infusion via an infusion pump. Always have a dedicated line for insulin.
Potassium: Do NOT add potassium to the first bag of IV fluids until renal function is confirmed (urine output) and a serum potassium level is obtained. Insulin drives potassium into cells, causing severe hypokalemia.
Blood Glucose Correction: The goal is to lower glucose by 50-75 mg/dL per hour. A too-rapid drop can cause cerebral edema. Once glucose reaches around 200 mg/dL, dextrose is often added to the IV fluids to prevent hypoglycemia while the insulin drip continues to clear ketones.
Nursing Procedure & Medication FlowManaging IV Insulin for DKA:
1. Obtain order for IV Regular Insulin infusion.
2. Prepare infusion: Add prescribed units (e.g., 100 units) to 100 mL of 0.9% NaCl (concentration = 1 unit/mL).
3. Label line clearly: "INSULIN INFUSION."
4. Program infusion pump to deliver prescribed rate (e.g., 7 units/hr for a 70 kg patient).
5. Check blood glucose every hour. The rate is titrated based on a pre-defined sliding scale or protocol.
6. When blood glucose approaches 200 mg/dL, anticipate adding D5 or D10 to the maintenance IV fluids.
7. Continue insulin drip until acidosis resolves (anion gap normalizes, serum ketones clear), NOT just until glucose normalizes.
A Word from Your Senior Nurse
"In the chaos of the ED, that fruity breath odor is your patient screaming for help on a metabolic level. Never ignore it. As a nurse, you are the first to detect these subtle yet critical signs. Connecting the pathophysiology (no insulin → fat burning → acids → Kussmaul breathing) to your assessment makes you proactive, not just reactive. When you study, don't just memorize 'DKA = fruity breath.' Understand the 'why' behind it. That deep understanding is what will allow you to act swiftly and confidently, not just on the NCLEX, but at the bedside where it truly matters."
핵심 개념
Diabetic Ketoacidosis — A life-threatening complication of diabetes characterized by hyperglycemia, ketosis, and metabolic acidosis due to severe insulin deficiency.
Kussmaul Respirations — Deep, rapid, and labored breathing pattern that is a compensatory mechanism for metabolic acidosis, commonly seen in DKA.
Metabolic Acidosis — A condition where the body produces too much acid or the kidneys cannot remove enough acid, resulting in a low blood pH (
Insulin Infusion — The continuous intravenous administration of regular insulin, which is the cornerstone of pharmacological treatment for DKA.
Sick Day Rules — Guidelines for patients with diabetes to follow during illness to prevent complications like DKA, including frequent glucose/ketone monitoring, maintaining fluid intake, and never stopping insulin.
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