A nurse is caring for a patient with type 1 diabetes mellitu… | 마이메르시 MyMerci
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문제

A nurse is caring for a patient with type 1 diabetes mellitus who presents to the emergency department. Which assessment finding would be the highest priority for immediate intervention?

A 28-year-old patient with type 1 diabetes mellitus is brought to the emergency department by family members who report the patient has been vomiting for the past 12 hours and appears confused.
해설
Blood glucose level of 450 mg/dL with positive urine ketones indicates diabetic ketoacidosis (DKA), which is a life-threatening emergency requiring immediate intervention to prevent coma and death.

심화 해설

Core Nursing Explanation Key Concept Analysis: This question tests the ability to prioritize nursing assessments in a patient with Type 1 Diabetes Mellitus (T1DM) presenting with vomiting and confusion. The core theme is recognizing the life-threatening complication of Diabetic Ketoacidosis (DKA). DKA is characterized by hyperglycemia, ketosis, and metabolic acidosis, leading to severe dehydration, electrolyte imbalance, and potential cerebral edema. The priority in nursing is to identify the most critical, life-threatening finding that requires immediate intervention to prevent deterioration.

Answer Rationale: Key Point! The correct answer is Blood glucose level of 450 mg/dL with positive urine ketones. This combination is the hallmark diagnostic finding for DKA. Hyperglycemia leads to osmotic diuresis, causing severe dehydration and electrolyte loss. Positive urine ketones confirm the body is breaking down fat for energy due to insulin deficiency, producing acidic ketone bodies that cause metabolic acidosis. This state is a medical emergency. While the other findings are concerning, they are often consequences of the underlying DKA process. Addressing the root cause (severe insulin deficiency and hyperglycemia) with IV fluids, insulin, and electrolyte replacement is the immediate priority to reverse the entire cascade.

Distractor Analysis:
Watch out for confusion! Option ② (Blood pressure of 90/60 mmHg with heart rate of 110 bpm) indicates hypovolemic shock secondary to severe dehydration from DKA. This is a critical finding, but it is a result of the hyperglycemia and osmotic diuresis. The priority intervention for shock (fluid resuscitation) is part of DKA management, but the diagnosis and definitive treatment (insulin) hinge on recognizing the hyperglycemia and ketosis first.
Option ③ (Temperature of 101.2°F with abdominal pain) suggests a possible infection, which is a common precipitating factor for DKA. However, fever and abdominal pain are also common symptoms of DKA itself. While infection needs to be treated, it does not take precedence over correcting the immediate, life-threatening metabolic derangement.
Option ④ (Respiratory rate of 28 breaths per minute with fruity breath odor) is a classic sign of DKA known as Kussmaul respirations. The body attempts to compensate for metabolic acidosis by exhaling more carbon dioxide (CO2). The fruity odor is from acetone, a type of ketone. This is a significant assessment finding that confirms acidosis but, again, is a compensatory mechanism for the primary problem of hyperglycemia and ketosis.

Related Concepts: The nursing process prioritization framework (e.g., ABCs—Airway, Breathing, Circulation) is crucial. In DKA, the immediate threat to life is from the metabolic crisis affecting all systems. Also, differentiate DKA from Hyperosmolar Hyperglycemic State (HHS), which features extreme hyperglycemia without significant ketosis, more common in Type 2 Diabetes. Concept Summary
ConceptKey FeaturesNursing Priority
Diabetic Ketoacidosis (DKA)Hyperglycemia (>250 mg/dL), Ketosis/Acidosis (low pH, positive serum/urine ketones), DehydrationImmediate IV fluids, IV insulin drip, electrolyte (especially potassium) replacement, identify & treat precipitating cause (e.g., infection).
Precipitating Factors for DKAInfection, missed insulin doses, new-onset diabetes, myocardial infarction (MI), stress.Thorough assessment for underlying cause (e.g., check for UTI, pneumonia).
Pathophysiology of DKAAbsolute insulin deficiency → Hyperglycemia → Osmotic diuresis → Dehydration/Electrolyte loss. Fat breakdown → Ketone production → Metabolic Acidosis.Understand the cascade to anticipate complications (shock, arrhythmias, cerebral edema).
Side-by-Side Comparison!
FeatureDiabetic Ketoacidosis (DKA)Hyperosmolar Hyperglycemic State (HHS)
Typical OnsetRapid (hours to days)Slower (days to weeks)
Blood GlucoseHigh (>250 mg/dL)Extremely High (>600 mg/dL)
Ketosis/AcidosisPresent (pH < 7.3)Absent or minimal
Mental StatusConfusion, drowsiness, comaProfound lethargy, coma more common
Primary Patient TypeType 1 Diabetes (can occur in Type 2)Type 2 Diabetes (especially elderly)
Key Mortality RiskCerebral edema (in treatment), acidosisSevere dehydration, thromboembolic events
Anatomy, Physiology & Pharmacology Points Physiology: Insulin allows glucose to enter cells. Without it, the body perceives starvation and breaks down fat, producing ketones (acetoacetate, beta-hydroxybutyrate, acetone). Ketones are strong acids that lower blood pH.
Pharmacology: Key Point! Treatment involves IV regular insulin drip to lower glucose and stop ketone production. Fluids (0.9% NaCl initially) correct dehydration. Potassium is added to IV fluids once urine output is confirmed, as insulin drives potassium into cells, causing dangerous hypokalemia. Memory Tips DKA Triad: Remember the "3 H's": Hyperglycemia, Hyperketonemia, Hyperosmolarity (with acidosis).
Kussmaul Respirations: Think "Kussmaul = Ketoacidosis." Deep, rapid breathing trying to "blow off" acid.
Priority Order: In DKA, think "Fluids before Insulin" to prevent shock from intravascular fluid shifts. High-Frequency NCLEX Topics DKA is a classic NCLEX topic. Expect questions on: 1) Identifying signs/symptoms (fruity breath, Kussmaul respirations, abdominal pain), 2) Prioritizing interventions (IV fluids first, then insulin), 3) Monitoring for complications during treatment (hypoglycemia, hypokalemia, cerebral edema), and 4) Patient education to prevent recurrence (sick-day rules, never skipping insulin). Watch Out for Question Variations! * Instead of "highest priority finding," the question may ask: "Which action should the nurse take first?" Answer: Initiate IV fluid resuscitation as ordered. * A question may focus on monitoring during treatment: "The nurse should monitor for which potential complication after initiating an insulin drip for DKA?" Answer: Hypokalemia. * A question may test patient education: "Which statement by a patient with T1DM indicates understanding of sick-day management?" Correct answer: "I will test my blood glucose every 3-4 hours and check my urine for ketones."

임상 시나리오

Nursing Clinical Practice Guide Clinical Scenario: You are the triage nurse in the ED. A young adult with known T1DM is lethargic, with dry mucous membranes, and a family member says, "They haven't been able to keep anything down, and their insulin pump might have failed."
Nursing Intervention Strategy: 1. Immediate Assessment (ABCDE): Ensure patent airway. Assess breathing pattern (look for Kussmaul). Attach cardiac monitor, obtain IV access x2 with large-bore catheters. 2. Diagnostic Focus: STAT point-of-care blood glucose and urine dip for ketones. Draw labs: BMP (Basic Metabolic Panel), venous blood gas, CBC. 3. Priority Interventions: * Fluid Resuscitation: Administer 1-2 liters of 0.9% NaCl over the first 1-2 hours as ordered. * Insulin Therapy: After fluids have started, initiate a continuous IV insulin drip (e.g., regular insulin) via an infusion pump. * Electrolyte Management: Once potassium level is known and urine output is adequate (>30 mL/hr), add potassium to maintenance IV fluids. 4. Ongoing Monitoring: Hourly vital signs, strict I&O, blood glucose checks hourly, neurologic checks every 1-2 hours (for cerebral edema).
Patient Safety and Precautions: Key Point! Never give a bolus of IV insulin in DKA; it must be a continuous infusion. Rapid drops in blood glucose or serum osmolality can cause cerebral edema. Monitor for signs: headache, change in mental status, bradycardia, hypertension. Nursing Procedure & Medication Flow IV Insulin Drip Protocol: 1. Confirm order: "Regular insulin ___ units in ___ mL NS, infuse at ___ units/hr." 2. Use an infusion pump. Label the line clearly: "INSULIN INFUSION." 3. Dual IV Line Setup: One line for insulin drip, a separate line for fluid/electrolyte replacement. This allows for independent rate adjustments. 4. Titration: The rate is titrated based on hourly blood glucose readings. Goal is to decrease glucose by 50-75 mg/dL per hour. 5. Transition to SubQ: When patient is stable, blood glucose < 200 mg/dL, and acidosis resolved, overlap subcutaneous insulin with the IV drip for 1-2 hours before discontinuing the IV. A Word from Your Senior Nurse "In the chaos of the ED, a patient with DKA can be overwhelming. Remember your ABCs and think pathophysiologically: they are dry, sour, and sweet (dehydrated, acidotic, hyperglycemic). Your rapid recognition and initiation of protocol-driven care literally saves lives. When you see that sky-high glucose with ketones, you know the game plan. On the NCLEX, they want to see that you can spot the fire (DKA) and not just the smoke (the symptoms it causes). Connect the dots from the lab value to the patient's condition, and you'll choose the right priority every time."

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