When a prolonged deceleration is identified, the nurse must act immediately to restore fetal oxygenation and prevent metabolic acidemia. The initial steps of intrauterine resuscitation are non-invasive and can be initiated by the bedside nurse without a direct provider order in most institutional protocols.
The primary goal is to maximize uterine blood flow. The left lateral position is the first-line intervention because it displaces the gravid uterus from the inferior vena cava and aorta, instantly improving maternal venous return and cardiac output. If the left lateral position does not resolve the deceleration, consider right lateral, hands-and-knees, or Trendelenburg positions to relieve potential umbilical cord compression.
Apply a non-rebreather face mask at 10 L/min to increase the maternal partial pressure of oxygen (PaO2). This creates a higher diffusion gradient across the placenta, maximizing oxygen delivery to the fetus. Oxygen therapy should be discontinued as soon as the fetal heart rate pattern normalizes to avoid potential fetal harm from prolonged hyperoxia.
Immediately discontinue any oxytocin infusion to reduce contraction frequency and strength, which can further compromise uteroplacental perfusion. If the deceleration persists, the provider may order a tocolytic agent (e.g., terbutaline 0.25 mg subcutaneously) to temporarily relax the uterus.
Assess maternal blood pressure. If hypotension is present, initiate an IV fluid bolus (e.g., 500-1000 mL of Lactated Ringer's solution) as ordered to rapidly expand intravascular volume and improve placental perfusion.
While initiating resuscitation, notify the obstetric provider immediately. If the prolonged deceleration does not resolve with these measures and fetal bradycardia persists, the team must prepare for an emergent cesarean delivery, as this indicates severe fetal compromise that cannot be managed conservatively.
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