A nurse is caring for a 68-year-old patient with diabetes me… | 마이메르시 MyMerci
Next Gen NCLEX
문제

A nurse is caring for a 68-year-old patient with diabetes mellitus type 2, chronic kidney disease stage 3, and heart failure with reduced ejection fraction. The patient reports feeling weak and nauseated, and recent laboratory results show elevated creatinine and potassium. Which nursing intervention should be prioritized?

해설
The patient's symptoms and comorbidities indicate a complex, urgent situation requiring immediate provider notification for assessment. Other options address specific issues but do not prioritize the need for comprehensive evaluation.

심화 해설

Core Nursing Explanation Key Concept Analysis: This question assesses the nurse's ability to prioritize care for a patient with multiple, interacting chronic conditions who is showing signs of acute deterioration. The core theme is Clinical Judgment and Prioritization in a complex medical-surgical scenario. The patient has diabetes mellitus type 2 (DM2), chronic kidney disease stage 3 (CKD stage 3), and heart failure with reduced ejection fraction (HFrEF). The new symptoms (weakness, nausea) and critical lab abnormalities (elevated creatinine and potassium) signal a potential acute kidney injury (AKI) on top of CKD and a dangerous electrolyte imbalance (Hyperkalemia). In HFrEF, the kidneys are already vulnerable due to reduced perfusion. Medications like ACE inhibitors (e.g., lisinopril) can further affect renal function and potassium levels. This combination creates a high-risk situation for life-threatening complications like severe hyperkalemia-induced cardiac arrhythmias. Answer Rationale: Key Point! The priority nursing intervention is to Immediately notify the healthcare provider. This is the correct answer because the nurse is identifying a cluster of data (subjective symptoms + objective lab values + complex medical history) that indicates a significant change in the patient's status requiring urgent medical evaluation and potential intervention. The nurse cannot independently manage this multifaceted problem; the provider needs to assess the need for interventions like ECG monitoring, medication adjustments (e.g., holding lisinopril or other nephrotoxic/renin-angiotensin-aldosterone system (RAAS) affecting drugs), managing hyperkalemia, and evaluating fluid status. This action aligns with the nursing process step of Implementation for a collaborative problem requiring physician/nurse practitioner intervention. Distractor Analysis:
Watch out for confusion! Option ②, "Hold the morning dose of lisinopril," is a tempting but incorrect first action. While lisinopril (an ACE inhibitor) can elevate creatinine and potassium, the nurse should not independently hold a critical cardiac and renal protective medication without an order, especially in a patient with HFrEF. The correct sequence is to notify the provider, who will then decide to adjust, hold, or continue the medication based on a full assessment. Holding it without notification could lead to destabilization of the patient's heart failure.
Option ①, "Administer prescribed insulin sliding scale," addresses hyperglycemia but is not the priority. The symptoms of weakness and nausea are more likely related to renal dysfunction and hyperkalemia than to uncomplicated hyperglycemia in this context. Furthermore, administering insulin could potentially worsen hypokalemia if the potassium is not critically high, but the immediate danger is from the reported elevated potassium.
Option ④, "Increase fluid restriction," is potentially harmful. The patient has symptoms (weakness, nausea) and lab findings (elevated creatinine) that could indicate Prerenal azotemia (reduced blood flow to kidneys), possibly from dehydration or worsening HF. Increasing fluid restriction without a provider's assessment could exacerbate renal impairment. Fluid management in a patient with both HF and CKD is delicate and requires medical guidance. Related Concepts: This scenario integrates concepts of Renal Failure, Electrolyte Imbalance (Hyperkalemia), Pharmacology (ACE inhibitors, diuretics), and The Cardiorenal Syndrome (where dysfunction of the heart and kidneys exacerbate each other). The nurse's role is to recognize the "red flags," initiate communication, and prepare for subsequent orders (e.g., ECG, potassium-binding resins, IV fluids or diuretics).
Concept Summary
ConceptKey Takeaway
Hyperkalemia in CKD & HFLife-threatening risk of cardiac arrhythmia. Symptoms: muscle weakness, nausea, ECG changes (peaked T waves).
ACE Inhibitors (e.g., Lisinopril)Renoprotective in CKD and cardioprotective in HF, but can cause Elevated serum creatinine and Hyperkalemia. Never hold without consulting provider.
Nursing Priority (ABCs)Airway, Breathing, Circulation. Hyperkalemia threatens Circulation (cardiac rhythm). Notification is the first step to secure it.
Acute Kidney Injury (AKI)Rapid decline in renal function. Manifested by rising creatinine. In this patient, could be prerenal (from HF) or intrinsic.

Side-by-Side Comparison!
ActionWhen it's the PRIORITYWhen it's NOT the priority (like in this question)
Notify ProviderFor new, critical lab values (K+ > 5.5 mEq/L, Cr rapidly rising), changes in neuro status, chest pain, etc.For routine, expected lab fluctuations within normal limits.
Hold a MedicationFor a clear, known severe allergic reaction or if a medication error is discovered.For a suspected side effect (like elevated Cr/K+ with ACEi) – notify first for an order.
Administer PRN MedFor acute, distressing symptoms per protocol (e.g., pain, nausea with antiemetic order).When symptoms are part of a larger, unstable clinical picture requiring diagnosis.

Anatomy, Physiology & Pharmacology Points
  • Kidney Function (Glomerular Filtration Rate - GFR): CKD stage 3 means GFR is 30-59 mL/min. The kidneys' ability to excrete potassium and creatinine is impaired.
  • Heart-Kidney Link (Cardiorenal Syndrome): In HFrEF, poor cardiac output reduces renal perfusion. The kidneys respond by activating the RAAS, which leads to vasoconstriction and sodium/water retention, worsening HF. ACE inhibitors block this system.
  • Lisinopril Mechanism: Inhibits Angiotensin-Converting Enzyme, preventing the formation of Angiotensin II. This lowers blood pressure, reduces cardiac afterload, and decreases proteinuria. A Key Point! side effect is reduced aldosterone secretion, which impairs potassium excretion in the kidneys.

Memory Tips
  • CRASH for Hyperkalemia Concerns: Creatinine up, Renal disease, ACE inhibitor use, Symptoms (weakness), High K+ = Notify STAT!
  • ACE-i & K+: Think "Avoid Concurrent Elevated K+" – ACE inhibitors can cause hyperKalemia.

High-Frequency NCLEX Topics The NCLEX-RN loves testing priority-setting in complex patients. This question combines three high-yield topics: Diabetes Management, Chronic Kidney Disease, and Heart Failure. Remember: When lab values are critically abnormal and the patient is symptomatic, notification and collaboration are almost always the first nursing action.
Watch Out for Question Variations!
  • Variation 1 (Focus on Action): "The nurse receives these lab results. What is the first action?" → Still Notify the provider.
  • Variation 2 (Focus on Assessment): "Before notifying the provider, what additional assessment is most critical?" → Answer: Obtain an ECG to assess for hyperkalemia-induced arrhythmias.
  • Variation 3 (Focus on Medication): "The provider orders sodium polystyrene sulfonate (Kayexalate). What is the nurse's priority action before administration?" → Answer: Assess bowel sounds and for presence of constipation (to prevent colonic necrosis).

임상 시나리오

Nursing Clinical Practice Guide Clinical Scenario: You are the day-shift nurse for Mr. Johnson, a 68-year-old with the listed comorbidities. During morning rounds, he says, "I just feel so weak today, and my stomach is upset." You review his morning labs: Serum Creatinine: 2.8 mg/dL (baseline 1.9), Potassium: 5.9 mEq/L. His vital signs are BP 150/92, HR 98, RR 22, O2 sat 94% on room air. Nursing Intervention Strategy: 1. Immediate Assessment: After noting labs, immediately assess the patient's cardiac rhythm (place on telemetry monitor if not already, or obtain a stat 12-lead ECG). Assess respiratory effort, lung sounds (for crackles indicating pulmonary edema), and mental status. 2. Priority Action: Call the healthcare provider (HCP) or use the secure messaging system. Use SBAR (Situation, Background, Assessment, Recommendation) communication: * S: "I'm calling about Mr. Johnson in room 402. He has new weakness and nausea, and his potassium is 5.9 and creatinine is 2.8, up from his baseline." * B: "He has a history of DM2, CKD stage 3, and HFrEF. His home meds include lisinopril 10mg daily, furosemide 40mg daily, and metformin." * A: "He is alert but weak. Vitals are stable but tachycardic. I'm about to get an ECG. No acute respiratory distress noted on auscultation yet." * R: "I recommend you evaluate him. Would you like me to hold the lisinopril and metformin this morning and obtain an ECG?" 3. Post-Notification Care: Based on HCP orders, prepare for interventions: administer ordered medications for hyperkalemia (e.g., IV calcium gluconate for membrane stabilization, insulin/glucose, albuterol nebulizer, sodium polystyrene sulfonate), ensure IV access, and monitor strict I&Os. Patient Safety and Precautions: * Do NOT administer potassium supplements, salt substitutes (high in KCl), or NSAIDs. * Hold medications per order: Commonly, the provider will order to hold metformin (risk of lactic acidosis in renal impairment), lisinopril, and possibly potassium-sparing diuretics. * Monitor for arrhythmias continuously. Peaked T-waves on ECG are an early sign of hyperkalemia.
Nursing Procedure & Medication Flow When Managing Hyperkalemia (Sample Orders): 1. ECG: Obtain stat. Document rhythm and any peaked T waves, widened QRS. 2. Calcium Gluconate (IV): Given to stabilize cardiac cell membranes. Administer slowly over 5-10 minutes via a patent IV. Monitor ECG during infusion. 3. Insulin (Regular) & Dextrose: Drives potassium into cells. Typically 10 units of regular insulin IV with 50 mL of 50% dextrose (D50). Monitor blood glucose closely every 30-60 minutes afterward to prevent hypoglycemia. 4. Albuterol Nebulizer: Also drives K+ into cells. Monitor for tachycardia. 5. Kayexalate (Sodium Polystyrene Sulfonate): Key Point! This is an exchange resin that removes potassium via the GI tract. It is given orally or as a retention enema. Critical Precaution: Ensure the patient is not constipated and has active bowel sounds before administration to reduce risk of bowel necrosis. Mix powder well in sorbitol or water as ordered.
A Word from Your Senior Nurse "In the real world, patients like Mr. Johnson are the rule, not the exception. They come with a whole 'laundry list' of problems. Your superpower as a nurse is seeing how those problems connect. That elevated potassium isn't just a number on a page; it's a direct threat to his heart rhythm, especially with his kidney and heart already struggling. Never be afraid to be the one who picks up the phone. Your vigilant assessment and timely communication are what stand between a stable chronic condition and a full-blown emergency. On the NCLEX and at the bedside, thinking 'big picture' and acting as the patient's advocate through collaboration will always serve you well."

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