Fine crackles throughout both lung fields with oxygen saturation of 88% indicates acute pulmonary edema, a life-threatening complication requiring immediate intervention. Other findings (elevated BP, creatinine, glucose) are concerning but not immediately life-threatening.
심화 해설
Core Nursing Explanation
This question tests the critical nursing skill of prioritization and clinical judgment in a complex, multi-problem patient. The core theme is identifying the most acute, life-threatening condition that requires immediate intervention, using the principles of ABCs (Airway, Breathing, Circulation) and Maslow's Hierarchy of Needs.
Key Concept Analysis
The patient has a complex history: Heart failure with preserved ejection fraction (HFpEF), COPD, and now hyperglycemia. The presenting symptoms (increasing shortness of breath, edema) suggest an acute exacerbation of heart failure. In HFpEF, the heart muscle is stiff, leading to high filling pressures. When the left ventricle cannot relax properly, pressure backs up into the left atrium and pulmonary veins, causing pulmonary congestion and fluid to leak into the alveoli (pulmonary edema). This directly impairs gas exchange, which is the most fundamental physiological need.
Answer Rationale
Key Point! Option ③ describes classic signs of acute pulmonary edema: fine crackles (rales) indicate fluid in the alveoli, and an oxygen saturation (SpO2) of 88% (Normal Value: >95%) signifies severe hypoxemia. This is an immediate threat to the patient's airway, breathing, and oxygenation—the top priority in any nursing assessment. This finding requires urgent actions like administering oxygen, elevating the head of the bed, notifying the provider, and preparing for possible diuretic administration.
Distractor Analysis
Watch out for confusion! It's easy to be distracted by other abnormal findings, but you must weigh their acuity.
① Blood pressure of 160/95 mmHg with a mild headache indicates hypertensive urgency. While concerning and needing management, it is not an immediate life threat like respiratory failure. The headache is mild, and there are no signs of end-organ damage (e.g., altered mental status, chest pain).
② An increase in serum creatinine from 1.8 to 2.1 mg/dL indicates worsening acute kidney injury (AKI), likely from reduced renal perfusion due to heart failure. This is a serious finding that requires investigation and intervention, but it does not pose an imminent threat to survival within the next minutes like respiratory failure does.
④ A blood glucose of 285 mg/dL with moderate ketones is significant. It suggests diabetic ketoacidosis (DKA) may be developing, especially in the context of acute illness (heart failure exacerbation is a stressor). However, "moderate" ketones without accompanying Kussmaul respirations, altered mental status, or severe metabolic acidosis (pH 50%) but the ventricle is stiff, impairing filling. In HFrEF, the ventricle is weak and can't pump effectively (EF
임상 시나리오
Nursing Clinical Practice Guide
Clinical Scenario: You are the nurse on a medical-surgical unit. Mr. Johnson, 72, admitted for heart failure management, calls you to his room. He is sitting upright in bed, struggling to speak in full sentences, visibly anxious, and his lips have a slight bluish tint. You immediately put on your stethoscope.
Nursing Intervention Strategy
1. Immediate Assessment (Within seconds):
* AIRWAY: Is it patent? Can he speak? (Yes, but with difficulty).
* BREATHING: Assess rate, rhythm, effort (Tachypnea, use of accessory muscles). Apply pulse oximeter – reads 88%.
* CIRCULATION: Quickly palpate radial pulse (rate, rhythm).
* Auscultation: Listen to lung bases – you hear fine, wet crackles (rales) throughout.
2. Immediate Interventions (Within 1-2 minutes):
* Positioning: Ensure he remains in High Fowler's position (head of bed at 90 degrees) to maximize lung expansion and reduce venous return (preload).
* Oxygen Therapy: Apply nasal cannula or non-rebreather mask as per order or protocol. Titrate to achieve SpO2 >92%. Key Point! In COPD patients, be cautious of high-flow oxygen potentially suppressing the hypoxic drive, but an SpO2 of 88% is an emergency – oxygenation takes priority.
* Notification: Call the provider STAT or activate the rapid response team (depending on facility policy) while initiating these measures.
* Reassurance: Stay calm. Tell the patient you are here to help and are getting the team. Anxiety worsens dyspnea.
3. Subsequent Actions & Monitoring:
* Obtain vital signs (BP, HR, RR).
* Prepare for medications: Have IV access ready. Anticipate orders for IV furosemide, nitroglycerin.
* Monitor for response: Reassess lung sounds, SpO2, respiratory effort, and urine output frequently (e.g., every 15-30 minutes initially).
Patient Safety and Precautions
* COPD and Oxygen: While "hypoxic drive" is a concern, never withhold oxygen from a severely hypoxemic patient (SpO2
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