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학습 자료 둘러보기 →A 2-year-old toddler with a history of ventricular septal defect (VSD) repair is admitted with signs of heart failure. While tachycardia, tachypnea, and gradual weight gain are expected findings in a compensated state, a sudden change in perfusion status demands immediate recognition and intervention.
The most critical assessment finding is the sudden onset of pallor with diaphoresis and weak peripheral pulses. This clinical triad signals a transition from compensated heart failure to low cardiac output syndrome or decompensated shock. Pallor and diaphoresis result from intense sympathetic nervous system activation to compensate for a falling cardiac output. Weak peripheral pulses directly reflect a diminished stroke volume and narrowing pulse pressure, indicating that the body's compensatory mechanisms are failing.
Immediate nursing actions include placing the patient on continuous cardiorespiratory monitoring and pulse oximetry, obtaining a full set of vital signs including blood pressure in all four extremities to assess for a narrow pulse pressure, and notifying the healthcare provider or rapid response team immediately. Prepare for potential interventions such as supplemental oxygen administration, intravenous fluid resuscitation with caution, and initiation or titration of inotropic support as ordered.
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