# An adult with diabetic ketoacidosis has dehydration and a low serum potassium level before insulin begins. Complete the initial treatment sequence.

> source: MyMerci (mymerci.kr)  
> url: https://mymerci.kr/pages/nclex_q.php?qn_id=547047&lang=en  
> language: en  
> subject: Fluid/Electrolyte Imbalances  
> category: PA

## Question

An adult with diabetic ketoacidosis has dehydration and a low serum potassium level before insulin begins. Complete the initial treatment sequence.

Initial treatment should include ::선택::oral fluid onlyroutine sodium bicarbonate as the first interventionintravenous crystalloid and assessment of the precipitating causefluid restriction. Because potassium is 3.2 mmol/L, the nurse should ::선택::start insulin immediately without potassiumignore the value because DKA always causes hyperkalemiagive a loop diureticreplace potassium and delay insulin until potassium rises above 3.5 mmol/L. Afterward, care includes ::선택::insulin with frequent glucose, ketone, electrolyte, renal, and acid-base monitoringinsulin without laboratory reassessmentsubcutaneous long-acting insulin as the only emergency treatmentstopping treatment when urine glucose becomes negative; when glucose falls below about 250 mg/dL, the team should ::선택::stop insulin even if ketosis persistsadd dextrose and continue insulin until ketoacidosis resolvesremove cardiac monitoringgive additional glucose-free fluid only.

## Explanation

The client meets current diagnostic criteria for diabetic ketoacidosis and has important total-body fluid and potassium deficits. Initial treatment includes intravenous crystalloid and evaluation of the precipitating cause. Because potassium is below 3.5 mmol/L, potassium replacement should begin and insulin should be delayed until potassium rises above that threshold to reduce the risk of lethal dysrhythmia and respiratory-muscle weakness. After insulin starts, glucose, beta-hydroxybutyrate, electrolytes, renal function, and venous pH require frequent monitoring. When glucose falls below about 250 mg/dL, dextrose is added while insulin continues until ketoacidosis resolves.

## In-depth explanation

Correct Answer
Begin fluid and potassium replacement, delay insulin for the initial hypokalemia, then treat with monitored insulin and dextrose as glucose falls.

Why It Fits
Insulin shifts potassium into cells and could worsen the existing potassium of 3.2 mmol/L to a life-threatening level.

Clinical Picture
Glucose correction alone does not define DKA resolution; ketonemia and acidosis must also resolve.

Memory Link
In DKA, fluid first and potassium decides when insulin is safe.

NCLEX Rule
If potassium is below 3.5 mmol/L, replace potassium and delay insulin until the level is safer, with continuous reassessment.

Why the Alternatives Fail
Immediate insulin despite hypokalemia, routine bicarbonate for this pH, or stopping insulin when glucose normalizes can cause preventable harm.

Sources
12024 Hyperglycemic Crises in Adults With Diabetes Consensus Report
2NCSBN 2026 NCLEX-RN Test Plan

## Clinical scenario

Emergency department
An adult with type 1 diabetes has vomiting, deep respirations, and dry mucous membranes. Glucose is 420 mg/dL, beta-hydroxybutyrate is 5.2 mmol/L, venous pH is 7.18, bicarbonate is 12 mmol/L, and potassium is 3.2 mmol/L. Cardiac monitoring is available.

## Key concepts

- **Diabetic ketoacidosis** — An acute metabolic emergency defined by diabetes or hyperglycemia, ketonemia, and metabolic acidosis.
- **Beta-hydroxybutyrate** — The predominant circulating ketone in DKA and a useful marker for diagnosis and resolution.
- **Total-body potassium deficit** — Loss of potassium from the body even when the initial serum value may appear normal or high.

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