# A nurse is assessing a patient with a complete C5 spinal cord injury who was admitted 2 hours ago following a motor vehicle accident. Which assessment finding would be the most critical indicator requiring immediate intervention?

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## 문제

A nurse is assessing a patient with a complete C5 spinal cord injury who was admitted 2 hours ago following a motor vehicle accident. Which assessment finding would be the most critical indicator requiring immediate intervention?

## 보기

1. Blood pressure 80/45 mmHg with heart rate 52 bpm **✔ 정답**
2. Absence of sensation below the nipple line
3. Inability to move lower extremities
4. Urinary retention with bladder distension

**정답: 1**

## 해설

Blood pressure 80/45 mmHg with heart rate 52 bpm indicates neurogenic shock, a life-threatening complication of acute spinal cord injury above T6 requiring immediate intervention. Other findings are expected deficits from the injury.

## 심화 해설

Understanding the Priority: Neurogenic Shock in Acute Spinal Cord Injury

This question tests your ability to recognize a life-threatening complication in a patient with an acute cervical spinal cord injury (SCI). The patient has a complete C5 injury, which means motor and sensory functions are lost below this level. However, the most immediate threat to life in the acute phase often stems from autonomic dysfunction, not the loss of motor or sensory function itself.

The correct answer is 1. Blood pressure 80/45 mmHg with heart rate 52 bpm. This combination of hypotension and bradycardia is the classic presentation of neurogenic shock. To understand why this is the priority, you must link the pathophysiology of the injury to the hemodynamic changes.

A complete SCI at the cervical level disrupts the descending sympathetic pathways from the brainstem. This causes a loss of sympathetic tone below the level of the injury. Without sympathetic outflow, there is unopposed parasympathetic activity via the vagus nerve, leading to two critical problems: severe vasodilation (causing a drop in systemic vascular resistance and blood pressure) and bradycardia. The definition of neurogenic shock in recent research aligns with this, characterizing it as hypotension, often defined as a systolic blood pressure (sBP) of ≤ 100 mmHg, in the setting of a SCI [1]. The patient's blood pressure of 80/45 mmHg is dangerously low and will lead to inadequate perfusion of vital organs, including the already vulnerable spinal cord, potentially worsening secondary injury.

Let's analyze why the other options are not the most critical immediate indicators requiring intervention, even though they are significant findings.

Analysis of Other Options

-   Option 2: Absence of sensation below the nipple line. The nipple line corresponds roughly to the T4 dermatome. For a complete C5 injury, a sensory level at T4 is an expected finding. The primary zone of injury is at C5, but the documented sensory level can often extend a few segments below the lesion due to spinal cord edema. This finding helps confirm the neurological level of injury but does not represent an immediately life-threatening change requiring emergent intervention over hemodynamic instability.

-   Option 3: Inability to move lower extremities. This is an expected finding in a complete C5 injury. The corticospinal tracts controlling motor function are interrupted, resulting in paralysis below the level of injury. While devastating, this is a direct consequence of the primary injury and is not the most critical finding in the hyperacute phase when compared to a state of shock.

-   Option 4: Urinary retention with bladder distension. This occurs due to the disruption of autonomic control to the bladder, leading to a flaccid, areflexic bladder. This is a common and important complication of SCI that requires intervention, such as catheterization, to prevent bladder damage. However, it is not as immediately life-threatening as the hemodynamic instability of neurogenic shock. The cardiovascular and hemodynamic consequences of autonomic dysfunction are associated with increased morbidity and mortality after SCI .

Clinical Reasoning and Immediate Action

Your clinical priority is grounded in the ABCs (Airway, Breathing, Circulation). Neurogenic shock is a form of distributive shock that directly compromises circulation. The hypotension is not due to volume loss but to profound vasodilation. The initial treatment focuses on restoring intravascular volume (with cautious fluid resuscitation) and, more importantly, providing vasopressor support to increase vascular tone. Simultaneously, the bradycardia may require atropine or pacing if it becomes symptomatic and severe. Recognizing this pattern of hypotension with bradycardia, as opposed to the tachycardia seen in hypovolemic or septic shock, is a key assessment skill for NCLEX-RN success. The other findings represent expected neurological deficits or secondary complications that are managed after the patient's circulatory status is stabilized.References (research sources)

- [1]Just a bit slow? Rethinking Neurogenic Shock – Insights from the TraumaRegister DGU®Research articleKamp O, Wolf M, Klenke N, Wolf RP, Vogel C, Becker L, Lefering R, Dudda M. (2026) · DOI: 10.21203/rs.3.rs-10203659/v1

## 임상 시나리오

Neurogenic Shock RecognitionAcute Cervical SCI Management
Suspect neurogenic shock with hypotension (SBP ≤ 100 mmHg) and bradycardia (HR < 60 bpm) in acute SCI above T6.

Pathophysiology involves loss of sympathetic tone, leading to vasodilation and unopposed vagal activity.

CautionDo not confuse with hypovolemic shock; fluid resuscitation alone may cause overload. Prioritize vasopressors and atropine for symptomatic bradycardia.

## 핵심 개념

- **Neurogenic Shock** — A distributive shock state resulting from loss of sympathetic tone after spinal cord injury, characterized by hypotension and bradycardia due to unopposed vagal activity.
- **Complete Spinal Cord Injury** — Total loss of motor and sensory function below the level of injury due to complete disruption of neural pathways.
- **Sympathetic Pathway Disruption** — Interruption of descending autonomic fibers in the spinal cord, leading to loss of vasomotor control and decreased systemic vascular resistance.
- **Vasodilation** — Widening of blood vessels due to loss of sympathetic tone, causing pooling of blood and severe hypotension.
- **Bradycardia** — Abnormally slow heart rate, often occurring in neurogenic shock due to unopposed parasympathetic stimulation via the vagus nerve.

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