Correct Answer: 2. Monitor blood pressure every 2 hours and implement fluid restriction
Understanding the Pathophysiology
Acute post-streptococcal glomerulonephritis (APSGN) is an immune-complex mediated disease, not a direct bacterial infection of the kidneys. Following a pharyngeal or skin infection with nephritogenic strains of group A beta-hemolytic Streptococcus, the body forms antigen-antibody complexes. These complexes deposit in the glomerular basement membrane, activating the complement system and causing a diffuse inflammatory response
[1]. This inflammation damages the glomeruli, the kidney's filtering units, leading to a predictable clinical picture.
The child's presentation—periorbital edema, hypertension (
140/90 mmHg), oliguria, proteinuria, and hematuria—is a classic constellation of symptoms resulting from this glomerular injury
[2]. The inflamed glomeruli have a reduced glomerular filtration rate (GFR). This means the kidneys cannot effectively filter waste products or, critically, regulate fluid and electrolyte balance. The result is a state of fluid volume overload. Sodium and water are retained, leading directly to the edema and, more dangerously, hypertension. The oliguria is a direct consequence of the decreased GFR, and the proteinuria and hematuria result from the damaged filtration membrane allowing proteins and red blood cells to leak into the urine.
Analyzing the Priority Intervention
The priority nursing intervention is driven by the most immediate life-threatening complication, which in this case is severe hypertension due to fluid volume overload. The goal of management is to support the child while the acute inflammation resolves, focusing on controlling the effects of the impaired renal function
[1].
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Option 2 is correct. Implementing strict fluid restriction and monitoring blood pressure (
BP) every
2 hours directly targets the core problem of fluid volume overload. Restricting intake prevents further worsening of the edema and hypertension, while frequent
BP monitoring is essential for early detection of hypertensive urgency or emergency, a serious complication of APSGN
[2]. This is a critical safety measure that directly manages the primary risk.
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Option 1 is incorrect and dangerous. Encouraging increased fluid intake would exacerbate the existing fluid volume overload. Since the kidneys are in a state of oliguria and cannot excrete the excess fluid, this intervention would worsen the edema and elevate the
BP further, potentially precipitating hypertensive crisis or pulmonary edema.
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Option 3 is incorrect. A high-protein diet is not indicated and could be harmful. The proteinuria is not due to a lack of protein intake but to a leaky glomerular membrane. Increasing dietary protein in the setting of a reduced
GFR would increase the load of nitrogenous waste products on the already damaged kidneys, which they are unable to clear effectively. Management typically involves a diet with controlled protein and sodium, not a high-protein diet.
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Option 4 is incorrect as a priority nursing intervention. While diuretic therapy is a common medical treatment to manage fluid overload and hypertension, it is not an independent nursing action. The nurse's priority is to assess and monitor the patient's response to therapy. Vigorous diuresis can lead to electrolyte imbalances and must be carefully managed by the healthcare provider. The nurse's role is to implement fluid restriction and provide the
BP monitoring data that guides the medical team's decision on whether and how aggressively to use diuretics
[1].
References (research sources)
- [1]
AsPNA Clinical Practice Guidelines for the management of infection-related glomerulonephritis.GuidelineMeena J, Sinha A, Krishnasamy S, Alba AA, Aziz MA, Begum A, Chan EY, Kari JA, Pattaragarn A, Perera I, Shen Q, Singh G, Bagga A, Glomerular Disease Working Group, Asian Pediatric Nephrology Association. (2026) · DOI: 10.1007/s00467-026-07146-4
- [2]
Acute Post-streptococcal Glomerulonephritis in a Pediatric Population: A Five-Year Retrospective Study.Research articleBajracharya P, Khadgi A, Shrestha S, Silwal R, Tandukar A. (2024) · DOI: 10.7759/cureus.56082