Primary Assessment Priority in Suspected Shoulder Dislocation
The most critical initial nursing assessment for a patient with a suspected shoulder dislocation is the
neurovascular status of the affected extremity. This is because the anatomical structures at risk during a glenohumeral dislocation are not limited to the joint itself. The
axillary artery and
brachial plexus are in immediate proximity to the humeral head, and displacement of the bone can lead to compression or direct injury of these vital structures
[1]. A delay in identifying a vascular insult, such as an axillary artery occlusion or pseudoaneurysm, directly increases the risk of limb-threatening ischemia, hemorrhage, and potential limb loss
[2].
While assessing the mechanism of injury, pain level, and range of motion are all components of a thorough secondary survey, they do not take precedence over evaluating for a time-sensitive neurovascular emergency. The presence of a distal pulse does not rule out a significant vascular injury. In cases of proximal humerus fracture-dislocation, bony impingement can compress the axillary artery while collateral circulation preserves a palpable radial pulse, creating a false sense of security
[1]. Similarly, a "floating shoulder" or infraclavicular injury can present with an axillary artery rupture that requires immediate multidisciplinary vascular repair alongside bone stabilization
[4]. The nurse must actively assess for the "six Ps" of compartment syndrome and acute arterial insufficiency (pain, pallor, pulselessness, paresthesia, poikilothermia, paralysis), as these findings indicate a progression from nerve compression to irreversible tissue damage.
The neurological assessment is equally paramount. The mechanism of a shoulder dislocation involves a risk for
brachial plexus injury (BPI), which can result in severe functional disability of the upper extremity
[3]. The axillary nerve is the most commonly injured structure, but more extensive traction or compression can cause a postganglionic lesion or a combined pre- and postganglionic injury
[3]. A focused motor and sensory examination comparing the affected limb to the unaffected side is essential to establish a baseline and determine if immediate nerve exploration or surgical reconstruction is indicated, particularly in a polytrauma context where life-saving interventions might delay a detailed neurological workup
[4]. The clinical manifestations of a BPI, such as loss of motor function in specific myotomes, can help localize the level of the lesion, but this assessment must be performed immediately to guide urgent treatment decisions
[3].
References (research sources)
- [1]
Radiological Diagnosis of Axillary Artery and Brachial Plexus Involvement in a Proximal Humerus Fracture-Dislocation: A Case Report.Case reportParkar N, Thangarajah T. (2025) · DOI: 10.7759/cureus.92425
- [2]
Axillary Artery Pseudoaneurysm Secondary to Septic Shoulder After Trauma: A Review of Incidence and Outcomes.Research articleKaur K, Mosa Basha B, Kaur H, Mohamed AM, Shetty S, Shehata Abdelmesih SR, Rashid N, Okoye J, Qandeel M, Ahmed K. (2025) · DOI: 10.7759/cureus.99982
- [3]
Brachial Plexus Injury in AdultsResearch articleHye Ran Park, Gwang Soo Lee, Il Sup Kim, Jae-Chil Chang (2017) · DOI: 10.21129/nerve.2017.3.1.1
- [4]
Challenges and Opportunities of Early Brachial Plexus Reconstruction in Polytrauma: Case Report and Review of the Literature.Case reportGiacalone M, Fiumedinisi F, Glaab R, Marti R, Plock JA, Frueh FS. (2026) · DOI: 10.3390/jcm15031300