A nurse is caring for a patient in cardiogenic shock followi… | 마이메르시 MyMerci
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문제

A nurse is caring for a patient in cardiogenic shock following an acute myocardial infarction. The patient's blood pressure is 80/50 mmHg, heart rate is 120 bpm, and urine output has decreased to 15 mL/hr. Which nursing intervention should be the highest priority?

The nurse must prioritize interventions for a patient experiencing cardiogenic shock with hemodynamic instability and decreased organ perfusion.
해설
In cardiogenic shock, the heart's pumping ability is severely compromised, leading to decreased cardiac output and tissue perfusion. The priority is monitoring for life-threatening complications like pulmonary edema while preparing for advanced interventions.

심화 해설

Core Nursing Explanation Key Concept Analysis: This question tests the priority nursing intervention for a patient in cardiogenic shock. The core pathophysiology is a profound decrease in cardiac output (CO) due to pump failure (from the myocardial infarction (MI)), leading to systemic hypoperfusion (shock) and a high risk of pulmonary edema as blood backs up into the lungs. The patient's vital signs (BP 80/50 mmHg, HR 120 bpm) and oliguria (urine output 15 mL/hr) confirm shock and end-organ damage.

Answer Rationale: Key Point! In cardiogenic shock, the failing left ventricle cannot eject blood effectively. This causes a rise in left ventricular end-diastolic pressure (LVEDP), which is transmitted back to the left atrium and pulmonary vasculature, leading to pulmonary congestion and edema. This is an imminent, life-threatening complication that can rapidly cause hypoxic respiratory failure. Therefore, the highest priority nursing action is vigilant monitoring for its signs (e.g., worsening dyspnea, crackles, pink frothy sputum, hypoxemia) and preparing for advanced support like mechanical ventilation, which may be necessary to maintain oxygenation.

Distractor Analysis: Watch out for confusion! Option ①, administering diuretics, is generally contraindicated in the early, unstable phase of cardiogenic shock. While fluid overload may be present, the primary problem is pump failure, not simply volume excess. Aggressive diuresis can further reduce preload and critically lower an already diminished cardiac output, worsening shock.
Option ②, increasing IV fluids, is the standard initial intervention for hypovolemic shock but is dangerous in cardiogenic shock. Adding more volume to a failing heart increases preload and myocardial workload, which can exacerbate pulmonary edema and further decrease cardiac output.
Option ③, the Trendelenburg position (head down), is an outdated and potentially harmful intervention for hypotension. It does not reliably improve blood pressure and can increase intracranial pressure and compromise respiratory function by forcing abdominal contents against the diaphragm, which is especially dangerous in a patient at risk for pulmonary edema.

Related Concepts: The management of cardiogenic shock focuses on improving cardiac output through inotropes (e.g., dobutamine) and vasopressors (e.g., norepinephrine), reducing cardiac workload, and ensuring adequate oxygenation. Interventions like intra-aortic balloon pump (IABP) counterpulsation may be used to support the heart. Nursing care revolves around continuous hemodynamic monitoring, managing medications, and preventing complications.

Concept Summary
ConceptExplanation
Cardiogenic ShockPump failure leading to inadequate cardiac output and tissue perfusion. Hallmark: Low CO with high systemic vascular resistance (SVR) and high pulmonary capillary wedge pressure (PCWP).
Priority ComplicationPulmonary edema due to left ventricular failure and pulmonary venous congestion.
Nursing PriorityMonitor respiratory status for signs of pulmonary edema and prepare for advanced airway/ventilatory support.
Contraindicated ActionsAggressive fluid boluses (increases preload stress) and routine diuretics in unstable phase (reduces preload excessively).

Side-by-Side Comparison!
Shock TypePrimary ProblemKey Hemodynamic ProfileFirst-Line Fluid Strategy
CardiogenicPump Failure (Heart)↓ CO, ↑ PCWP, ↑ SVRFluid RESTRICTION/Cautious
HypovolemicVolume Loss (Tank)↓ CO, ↓ PCWP, ↑ SVRAGGRESSIVE Fluid Resuscitation
Septic (Distributive)Vasodilation (Pipes)↑ CO (early), ↓ PCWP, ↓ SVRAggressive Fluids followed by Vasopressors

Anatomy, Physiology & Pharmacology Points The left ventricle's failure increases pressure in the pulmonary circulation. When hydrostatic pressure in pulmonary capillaries exceeds the oncotic pressure that holds fluid in the vessels, fluid leaks into the alveoli, causing pulmonary edema. First-line drugs include dobutamine (inotrope to improve contractility) and norepinephrine (vasopressor to increase SVR and perfusion pressure), often given via central line.

Memory Tips Think "Pump problem = Protect from Pulmonary edema." Remember the rule: NO "F" for the Failing Heart – No aggressive Fluids. In cardiogenic shock, you support the pump, you don't fill the tank.

High-Frequency NCLEX Topics Shock is a high-yield topic. NCLEX loves to test your ability to differentiate the type of shock based on symptoms and to identify the priority intervention, which is often opposite between hypovolemic and cardiogenic shock. Recognizing contraindicated actions is just as important as knowing the correct ones.

Watch Out for Question Variations! This concept can be tested by: 1) Asking for the contraindicated intervention (e.g., "Which action should the nurse avoid?" Answer: Rapid IV fluid bolus). 2) Presenting a patient with crackles and hypotension and asking for the likely shock type (Cardiogenic). 3) Shifting to medication priority (e.g., "Which IV medication should the nurse prepare to administer first?").

임상 시나리오

Nursing Clinical Practice Guide Clinical Scenario: You are the nurse in the Cardiac Intensive Care Unit (CICU). Mr. Johnson, 68, was admitted with an acute anterior MI. He now has cool, clammy skin, is increasingly restless and dyspneic, and his telemetry shows sinus tachycardia. His SpO2 is dropping to 88% on 4L nasal cannula.

Nursing Intervention Strategy: 1. Assessment: Immediate focused assessment on respiratory status. Auscultate lung fields bilaterally (listening for crackles/wheezes), assess work of breathing, check SpO2 continuously. Monitor hemodynamics from arterial and central lines if present (BP, heart rate, cardiac output/index, PCWP). 2. Action: Elevate the head of the bed (High-Fowler's position) to improve ventilation and reduce venous return to the heart. Apply supplemental oxygen as ordered and prepare for non-rebreather mask or BiPAP/mechanical ventilation. Notify the provider STAT with your assessment findings. 3. Medication & Care: Administer inotropic/vasoactive drips as ordered via an infusion pump, using a central line. Titrate carefully based on parameters. Restrict IV fluids as ordered. Maintain strict I&O, monitoring that urine output remains above 0.5 mL/kg/hr.

Patient Safety and Precautions: Never leave this patient unattended. Rapid deterioration is possible. Ensure all emergency equipment (crash cart, intubation tray, suction) is at the bedside. When administering potent vasoactive drugs, always use a double-check process for dose and line to prevent medication errors.

Nursing Procedure & Medication Flow Managing Vasoactive Drips: 1. Verify order, drug, dose, and patient identity with another nurse. 2. Prepare infusion using a central venous line (preferred for vesicant drugs). 3. Label the line clearly: "VASOACTIVE – DO NOT BOLUS". 4. Program the infusion pump with the correct rate (usually mL/hr or mcg/kg/min). 5. Monitor blood pressure and heart rate every 5-15 minutes during titration. Never stop the infusion abruptly. 6. Have a second IV line with a non-drug fluid running to keep the vein open (KVO) in case the primary line fails.

A Word from Your Senior Nurse "In the chaos of a crashing patient, your ABCs (Airway, Breathing, Circulation) are your anchor. For this cardiogenic shock patient, the 'B' – Breathing – is the most immediate threat. Your keen assessment for the crackles of pulmonary edema and your swift action to optimize oxygenation can be the difference between stabilizing the patient and a code blue. On the NCLEX and at the bedside, thinking pathophysiologically – 'The pump is failing, so blood is backing up into the lungs' – will always guide you to the right priority. You are the eyes, ears, and advocate for your patient when they are at their most vulnerable."

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