A nurse is developing a care plan for a client diagnosed wit… | 마이메르시 MyMerci
Adult Health
문제

A nurse is developing a care plan for a client diagnosed with syndrome of inappropriate antidiuretic hormone secretion (SIADH) following a head injury. Which intervention should the nurse include in the plan of care?

해설
SIADH causes excessive ADH secretion, leading to water retention and dilutional hyponatremia. The priority intervention is fluid restriction to prevent further water retention and worsening hyponatremia.

심화 해설

Core Nursing Explanation This question tests the understanding of the pathophysiology of Syndrome of Inappropriate Antidiuretic Hormone (SIADH) and the corresponding priority nursing intervention. SIADH is characterized by the inappropriate, continuous secretion of Antidiuretic Hormone (ADH or Vasopressin) from the posterior pituitary gland, regardless of the body's actual serum osmolality or fluid volume. Key Concept Analysis The core problem in SIADH is Key Point! water retention. Excessive ADH acts on the kidneys, increasing water reabsorption in the collecting ducts. This leads to: 1. Expansion of extracellular fluid volume (but often without visible edema because the excess water distributes evenly). 2. Dilutional Hyponatremia: The retained water dilutes the sodium in the blood, causing serum sodium levels to fall (Na+ < 135 mEq/L). 3. Watch out for confusion! The body's total sodium content is usually normal, but it's *diluted* by excess water. Answer Rationale The cornerstone of medical management for SIADH is Fluid Restriction. By limiting total fluid intake (typically to 800-1000 mL/day, as indicated in the correct answer), you reduce the amount of "free water" available for the kidneys to retain under the influence of ADH. This helps to: * Stop the worsening of hyponatremia. * Allow the kidneys to excrete the excess water gradually. * Raise the serum sodium concentration toward the normal range (135-145 mEq/L). Distractor Analysis * ② Encourage increased sodium intake: This is incorrect and potentially dangerous. Adding dietary sodium without correcting the underlying water excess is ineffective and can be harmful. In some cases, severe symptomatic hyponatremia may be treated with hypertonic saline (3% NaCl) IV under strict monitoring, but this is a high-risk intervention, not a dietary change. * ③ Monitor for signs of hypernatremia: This shows a fundamental misunderstanding. SIADH causes Hyponatremia. Monitoring should be for signs of hyponatremia, such as headache, confusion, lethargy, seizures, and muscle cramps. * ④ Administer diuretics as prescribed: While loop diuretics (like furosemide) are sometimes used in conjunction with saline for severe cases, they are not a first-line or standalone treatment. Administering diuretics alone can exacerbate fluid and electrolyte losses and worsen the patient's condition. The primary intervention is always fluid restriction. Related Concepts Understanding SIADH is incomplete without contrasting it with its opposite: Diabetes Insipidus (DI). In DI, there is a deficiency of ADH (or kidney resistance to it), leading to profound water loss, hypernatremia, and dehydration. The treatment is the opposite: fluid replacement and administration of synthetic ADH (desmopressin).
Concept Summary
ConceptPathophysiologyKey Lab FindingPriority Intervention
SIADHInappropriate ADH excess → Water retentionDilutional HyponatremiaFluid Restriction
Diabetes Insipidus (DI)ADH deficiency → Profuse water lossHypernatremiaFluid Replacement / Desmopressin

Side-by-Side Comparison!
FeatureSIADH (Too much ADH)Diabetes Insipidus (Too little/no ADH effect)
Urine OutputLow (concentrated, high specific gravity >1.030)Very High (dilute, low specific gravity

임상 시나리오

Nursing Clinical Practice Guide Clinical Scenario: You are caring for Mr. Johnson, a 58-year-old who sustained a traumatic brain injury in a fall. Three days post-injury, he becomes increasingly lethargic and confused. His urine output has decreased significantly, and his lab work shows: Sodium 130 mEq/L, Serum Osmolality low, Urine Osmolality high. The physician diagnoses SIADH and writes an order: "Restrict total fluids to 1000 mL/24 hours." Nursing Intervention Strategy 1. Assessment: Perform frequent neurological checks (every 1-2 hours initially) using a tool like the Glasgow Coma Scale (GCS). Monitor for subtle changes: increased headache, disorientation, muscle twitching, or seizure activity. Accurately measure and record all Intake and Output (I&O). 2. Planning & Implementation (Fluid Restriction): * Calculate the hourly fluid allowance (1000 mL / 24 hrs ≈ 42 mL/hr). * Collaborate with dietary services to provide a fluid-restricted meal tray. * Key Point! Educate the patient and family on the critical reason for fluid restriction. Explain that even ice chips, gelatin, and ice cream count as fluid. * Provide meticulous oral care every 2 hours with mouthwash or lemon-glycerin swabs to alleviate thirst and dry mouth. * Schedule fluids strategically throughout the day to help the patient cope. * Administer all IV medications in the smallest volume of fluid possible (e.g., saline flushes count toward intake). 3. Evaluation: Monitor serum sodium levels daily. The goal is a slow, gradual increase (no more than 6-8 mEq/L in the first 24 hours). Improvement in neurological status is the best clinical indicator of effective treatment. Patient Safety and Precautions * Seizure Precautions: Due to the risk of severe hyponatremia leading to seizures, implement bed alarms, padded side rails, and have suction equipment available. * Medication Caution: If hypertonic saline (3% NaCl) is ordered, it must be administered via an IV infusion pump on a dedicated line, with frequent serum sodium checks (every 2-4 hours initially). * Fall Risk: The patient's confusion and lethargy increase fall risk. Keep the bed in low position, and ensure call light is within reach.
Nursing Procedure & Medication Flow Managing Strict Fluid Restriction: 1. Label the patient's room and chart with "Fluid Restriction" alert. 2. Use a graduated pitcher for water at the bedside to visually track intake. 3. Record the volume of all oral, IV, tube feed, and flush fluids in the I&O sheet. 4. At the end of each shift, total the intake and compare it to the prescribed limit. Report significant variances. A Word from Your Senior Nurse "SIADH is a perfect example of how a tiny hormone can cause big problems. In neuro units, we see it often after head injuries. Remember, your most important tools are your eyes and your assessment skills. That slight increase in confusion or new headache could be the first sign of dropping sodium. Fluid restriction seems simple, but it's challenging for patients. Your empathy and creative nursing care—like fantastic oral care and scheduling favorite fluids—make all the difference. You're not just following an order; you're protecting their brain from swelling and seizures. Connect the dots: head injury → possible pituitary disruption → SIADH → hyponatremia → neuro changes. That's the kind of critical thinking that saves lives and ace's the NCLEX!"

핵심 개념

  • Syndrome of Inappropriate Antidiuretic Hormone — A condition of excessive, non-suppressible secretion of antidiuretic hormone (ADH/vasopressin) leading to water retention, dilutional hyponatremia, and concentrated urine.
  • Dilutional Hyponatremia — Low serum sodium concentration (
  • Fluid Restriction — A therapeutic intervention limiting total daily fluid intake (often to 800-1000 mL) to correct or prevent worsening of fluid volume excess and hyponatremia, as in SIADH.
  • Antidiuretic Hormone (ADH / Vasopressin) — A hormone produced in the hypothalamus and released from the posterior pituitary that acts on the kidneys to increase water reabsorption, concentrating urine and conserving body water.
  • Diabetes Insipidus — A disorder characterized by a deficiency of ADH or renal resistance to its effects, resulting in the excretion of large volumes of dilute urine, intense thirst (polydipsia), and hypernatremia.

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