Quick answerPulmonary embolism. Take action: apply oxygen to a saturation above 90% and start the IV heparin protocol. Monitor: oxygen saturation and anti-factor Xa or aPTT level.
Why this is correctOrthopedic surgery, immobility, and missed enoxaparin create the Virchow triad; the enlarged right calf suggests the source DVT. Hypoxemia with clear lungs means a perfusion problem rather than an airway or alveolar one. The client is hypotensive-borderline but not in shock, so anticoagulation is the standard therapy; heparin dosing is titrated to anti-Xa or aPTT results, and oxygenation shows whether gas exchange is improving.
Easy analogy or mental pictureA clot has plugged a pipe to the lungs: air still reaches the rooms (clear lungs) but blood cannot pick it up. Oxygen raises what reaches the blood; heparin stops the plug from growing.
Memory hookCLEAR lungs + LOW sat + SUDDEN = clot: O2 + HEPARIN; watch SAT and the heparin level.
NCLEX decision ruleWhen hypoxemia is out of proportion to lung sounds after surgery, think perfusion; treat hypoxemia first, start the definitive therapy, and monitor both oxygenation and the drug's therapeutic level.
Why the other choices are wrongFat embolism typically appears 24–72 hours after long-bone fracture or surgery with petechiae and confusion, which are absent. Acute coronary syndrome is less likely with pleuritic pain and an ECG without ST elevation, so nitroglycerin is not indicated. Pneumonia would bring fever, sputum, and focal findings; incentive spirometry and WBC count track atelectasis or infection. A rapid large-volume bolus can worsen right ventricular strain in a client who is not in shock. Incision drainage tracks surgical-site bleeding rather than the embolism.
References
1NHLBI: Venous ThromboembolismRisk factors, signs, and treatment of pulmonary embolism after surgery
2DailyMed: Heparin Sodium LabelsHeparin dosing and laboratory monitoring with aPTT or anti-factor Xa