A client with an acute COPD exacerbation has an oxygen satur… | MyMerci
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Medication AdministrationPPT
Question
A client with an acute COPD exacerbation has an oxygen saturation of 82% on room air. Which initial oxygen plan is most appropriate?
1Withhold oxygen until arterial blood gas results confirm the degree of hypoxemia.
2Apply uncontrolled high-flow oxygen and maintain saturation above 98% throughout treatment.
3Start noninvasive ventilation at 100% oxygen without first assessing the response to controlled oxygen.
4Start controlled oxygen, titrate to 88% to 92%, and reassess clinical status and blood gases.✓ Correct answer
Explanation
A client with a COPD exacerbation and severe hypoxemia needs oxygen promptly. Controlled oxygen is titrated to a target saturation of 88% to 92% while the nurse reassesses respiratory status and obtains blood gases to identify carbon dioxide retention or acidosis. Oxygen should not be withheld. Uncontrolled oxygen to a saturation above 98% can worsen hypercapnia in susceptible clients, and ventilatory support is escalated according to clinical and blood-gas findings.
Clinical reasoning Hypoxemia is immediately dangerous, but excessive oxygen can increase carbon dioxide retention during some COPD exacerbations. The safe approach is prompt, controlled titration with reassessment.
Decision rule Target 88% to 92% during an acute COPD exacerbation and use blood gases and clinical response to guide further respiratory support.
Clinical scenario
Scenario 71-year-old male with severe COPD and chronic CO2 retention (baseline PaCO2 58). ED presentation: dyspnea, RR 28, SpO2 86% on room air, alert, mild lethargy.
Key concepts
Controlled oxygen therapy — Oxygen delivered and adjusted to a defined saturation target rather than at an uncontrolled high concentration.
Oxygen-induced hypercapnia — A rise in carbon dioxide that can occur in susceptible clients with COPD when excessive oxygen alters ventilation-perfusion balance and other physiologic mechanisms.
CO2 narcosis — CNS depression from rising PaCO2; presents as somnolence and confusion after high-flow O2.