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Therapeutic Management | 마이메르시 MyMerci
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Therapeutic Management

NCLEX Review Guide: Cardiovascular Therapeutic Management

Cardiovascular Medications

ACE Inhibitors & ARBs

  • ACE inhibitors (lisinopril, enalapril) block conversion of angiotensin I to angiotensin II, reducing blood pressure and cardiac workload. Monitor for hyperkalemia and angioedema.
  • ARBs (losartan, valsartan) block angiotensin II receptors with similar effects but lower risk of cough and angioedema compared to ACE inhibitors.
Memory Aid: "ACE the BP" - ACE inhibitors reduce blood pressure by blocking the conversion process

Beta Blockers

  • Metoprolol, atenolol, carvedilol block beta-adrenergic receptors, reducing heart rate, contractility, and blood pressure. Never stop abruptly - risk of rebound hypertension.
  • Contraindicated in severe asthma, COPD, and heart block. Monitor for bradycardia and hypotension.

Calcium Channel Blockers

  • Amlodipine, nifedipine (dihydropyridines) primarily cause vasodilation. Diltiazem, verapamil (non-dihydropyridines) affect heart rate and contractility.
  • Monitor for peripheral edema, constipation, and gingival hyperplasia. Avoid grapefruit juice with certain formulations.

Key Points

  • Always check apical pulse before giving cardiac medications
  • Monitor electrolytes, especially potassium with ACE inhibitors and diuretics
  • Teach patients to change positions slowly to prevent orthostatic hypotension

Heart Failure Management

Medication Therapy

  1. Diuretics (furosemide) reduce preload by decreasing fluid volume. Monitor for hypokalemia, hyponatremia, and dehydration.
  2. Digoxin increases contractility and decreases heart rate. Therapeutic level: 0.5-2.0 ng/mL. Toxicity signs: nausea, visual changes, arrhythmias.
  3. Aldosterone antagonists (spironolactone) are potassium-sparing diuretics that improve survival in heart failure.
Clinical Scenario: Patient with heart failure on furosemide reports muscle cramps and weakness. Priority action: Check serum potassium level and assess for hypokalemia.

Non-Pharmacological Management

  • Sodium restriction (2-3g daily) and fluid restriction (1.5-2L daily) reduce fluid retention and cardiac workload.
  • Daily weights at same time with same scale - report weight gain >2-3 lbs in 24 hours or >5 lbs in week.
  • Activity modification with gradual increase in exercise tolerance and energy conservation techniques.

Key Points

  • Position patients in high Fowler's to reduce preload and improve breathing
  • Monitor I&O closely and assess for signs of fluid overload
  • Teach patients to recognize early signs of worsening heart failure

Commonly Confused Concepts

Concept Key Difference NCLEX Tip
Preload vs Afterload Preload = venous return; Afterload = arterial resistance Preload = "before" the heart contracts
Systolic vs Diastolic HF Systolic = pumping problem; Diastolic = filling problem Systolic = "squeeze"; Diastolic = "stretch"
Stable vs Unstable Angina Stable = predictable; Unstable = unpredictable/worsening Unstable = "unpredictable" and urgent
Memory Aid for Heart Sounds:
S1 = "lub" (tricuspid/mitral closure)
S2 = "dub" (aortic/pulmonic closure)
S3 = gallop (heart failure)
S4 = atrial gallop (hypertension)

Common Pitfalls & Quick Checks

Common Pitfalls:
  • Forgetting to check apical pulse before cardiac medications
  • Not recognizing digoxin toxicity early signs
  • Confusing signs of right vs left heart failure
  • Missing orthostatic vital sign changes
Quick Check - Self Assessment: Can you identify therapeutic ranges for digoxin?
Do you know the difference between ACE inhibitors and ARBs?
Can you list signs of heart failure exacerbation?
Do you understand preload vs afterload concepts?

Remember: You've got this! Focus on understanding the "why" behind each intervention. Cardiovascular nursing requires critical thinking about hemodynamics and patient safety. Trust your knowledge and clinical judgment!

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